Blushing - Hyperpyrexia, Red Burning Face Symptoms and Treatment
Facial blushing is
often mis-diagnosed as rosacea. A
red face is a typical manifestation of this contdition. Fear
of social situations can bring on blushing and is known as erythrophobia. Blushing
commonly originates at the upper chest or base of the neck and
extends up to the forehead, ears and can even spread down to include
the trunk and legs. Facial sweat often accompanies blushing and
feelings of burning or tingling.
(feelings of burning or tingling) is commonly associated with
blushing and is actually much more debilitating to the individual
than the mere appearance of blushing on the skin. This condition
of hyperpyrexia associated with facial blushing is little understood
and not typically recognized by many physicians. Functional impairment
arises from the intense "burning" sensation that becomes
so uncomfortable that the individual has to actually stop what
they are doing because of the severe inability to concentrate
or follow through with the task at hand.
Many patients have
described the feeling they experience from hyperpyrexia as "hot
flash or burning." This sensation envelops their face/head/neck
to such an extent that they seek seclusion to "cool off".
- BeforeMicro ETS™ RESULTS MAY VARY FROM PERSON TO PERSON.
Blushing - After Micro ETS™ (Click to Enlarge) RESULTS MAY VARY FROM PERSON
are seldom helpful for this intense "hot or burning" feeling.
Beta blockers (i.e.: Propranolol are commonly used but to little
for Facial Blushing Video
Even when the individual is relaxed,
blushing episodes may occur. Sometimes excess sweating of the scalp
and face is associated with blushing / hyperpyrexia attacks. This
condition often causes the individual to become self-conscious,
nonproductive at work or school during the episodes, and to develop
a low self esteem. Some patients that Dr.
Nielson has treated have actually become so severely dysfunctional
that they quit work and would move from job to job.
Many physicians don't recognize
that these blushing episodes are commonly associated with hyperpyrexia
of the face which is actually what is so debilitating and causes
the severe functional impairment. The combination of facial blushing
with the warm/burning sensation of the head commonly makes the
individual extremely anxious and quite uncomfortable. This uncomfortable
feeling can be so intense that the individual can lose all ability
to concentrate and function properly.
Medications commonly used to
prevent or control blushing include beta blockers (Inderal) and
anxiolytics (sedatives). These medications are usually of limited
benefit in reducing blushing intensity and frequency of episodes.
Blushing - Before Micro
ETS RESULTS MAY VARY FROM PERSON TO PERSON.
Micro ETS RESULTS MAY VARY FROM PERSON TO PERSON.
Effective treatment for facial blushing
with hyperpyrexia is Micro ETS of T2.Precise
division of the sympathetic nerve exactly where it crosses the
upper edge of the second rib and any kuntz nerve branches
present crossing the second thoracic rib as well. Dividing all
kuntz nerves as they cross the second thoracic rib is essential
for successful treatment of facial blushing.
Persistent facial blushing after
sympathectomy can occur if any kuntz nerve that crosses the second
thoracic rib is left intact, and can therefore continue to carry
nerve signals to the face.
In the many redo's Dr.
Nielson has performed for persistent facial blushing, he
has found that the most common reason for persistent blushing
has been missed Kuntz nerves crossing the second thoracic rib.
He has also found, less commonly, the sympathetic nerve had been
divided at the T3 level by mistake rather than the T2 level.
T2 Sympathetic Innervation
to The Sweat Glands of the Face
Video - Student's Suicide Shines Light on 'Chronic Blushing'
Over several years of experience
in treating patients with recurrent and/or persistent sweating
of the face after undergoing T2 sympathectomy,Dr.
Nielson has found that persistence of any sympathetic nerve innervation
across the second rib level, just above the T2 ganglion, plays
a significant role in persistent sweating conditions of the face
after undergoing a T2. T3, or T4 sympathectomy.
is apparent in some patients that there are neuronal contributions
from lower levels such as the T3 that pass up over the second
rib level on their way to the face that participate in the sweating
symptoms of the face. Some physicians misunderstand the sympathetic
nerve innervation of the face and believe in order to successfully
treat facial sweating it is important to cut the sympathetic nerve
at the T1 level or above, thereby causing the dreaded Horner’s
Syndrome. In Dr.
Nielson's experience, he has found this not to be the case.
In summary, for successful
treatment of facial sweating, it is imperative that all sympathetic
nerve innervation crossing the second rib level be divided as opposed
to clamped or having lower levels cut or clamped. Also, accessory
nerve branch pathways bypassing the T2 ganglion can or may contribute
to persistent facial symptoms.